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Through the nAChRs in brain reward circuits, nicotine alters dopaminergic (DA) neuronal function in the ventral tegmental area (VTA) and increases dopamine release from VTA to nuclear accumbens (NA), which leads to nicotine reward, tolerance and dependence

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(52) also showed that autophagy activity was blocked in traumatic HF in AA patients

[DOI] [PMC free article] [PubMed] [Google Scholar] 138

Then, we applied our previously proposed drug repositioning method which aims to repurpose useful drugs for targeting a specific gene based on the similarity analysis of the perturbations induced by the target gene knockdown and candidate drug treatment on the available human cell lines [13, 18]